Endotoxemia as produced in rats by i.p. injection of LPS. in the present study, bile flow rate, biliary excretion of GSH and lysosomal glycosidases were measured. In addition to the meaurement of LPO, NO and some antioxidant enzymes activities in the liver and eythrocytes lysate. The results foud that LPS caused a sgnificant increase in AST,ALT, GGT, LPO and NO. However, bile flw rate and the biliary excretion of GSH andlysosomal glycoidases and antioxdant enzymes activiies were decreased.The study indicated that LPS induced hepatic injury in rats by increased release of ROS which is consumed endogenous antioxidant.

The experiment was designed to investigate the role of tannic acid as antioxidant in reduction of the oxidtive damage of ALCL3 in the brin of rat The obtained biochemical and histological results documentd that tannic acid has a prophylactic role in reducing the oxidaive amage in the bain tissues that caused by ALCL3 exposure for80 days in drinking water.

The experiment was designed to investigate the role of tannic acid as antioxidant in reduction of the oxidtive damage of ALCL3 in the brin of rat The obtained biochemical and histological results documentd that tannic acid has a prophylactic role in reducing the oxidaive amage in the bain tissues that caused by ALCL3 exposure for80 days in drinking water.

The experiment was designed to investigate the role of tannic acid as antioxidant in reduction of the oxidtive damage of ALCL3 in the brin of rat The obtained biochemical and histological results documentd that tannic acid has a prophylactic role in reducing the oxidaive amage in the bain tissues that caused by ALCL3 exposure for80 days in drinking water.

The experiment was designed to investigate the role of tannic acid as antioxidant in reduction of the oxidtive damage of ALCL3 in the brin of rat The obtained biochemical and histological results documentd that tannic acid has a prophylactic role in reducing the oxidaive amage in the bain tissues that caused by ALCL3 exposure for80 days in drinking water.

Pospectiv studies suggest that tea may protect against oxidative stress that induced tissue damage. A potential mechanism fr such effect y polyphenolc antioxidant tannic acid derived from tea was evaluated in tis study. Treatment of rats with ALCL3 for 80 days caused moderate toxicity on liver, kidney and splen as evidenced by increased LPO& No and a reduction of SOD,CAT, GST, GSH nd vitamin E.As well as histopathological changes.However improvement was noticed in the rats treated with tannic acid.

Pospectiv studies suggest that tea may protect against oxidative stress that induced tissue damage. A potential mechanism fr such effect y polyphenolc antioxidant tannic acid derived from tea was evaluated in tis study. Treatment of rats with ALCL3 for 80 days caused moderate toxicity on liver, kidney and splen as evidenced by increased LPO& No and a reduction of SOD,CAT, GST, GSH nd vitamin E.As well as histopathological changes.However improvement was noticed in the rats treated with tannic acid.

Pospectiv studies suggest that tea may protect against oxidative stress that induced tissue damage. A potential mechanism fr such effect y polyphenolc antioxidant tannic acid derived from tea was evaluated in tis study. Treatment of rats with ALCL3 for 80 days caused moderate toxicity on liver, kidney and splen as evidenced by increased LPO& No and a reduction of SOD,CAT, GST, GSH nd vitamin E.As well as histopathological changes.However improvement was noticed in the rats treated with tannic acid.

Pospectiv studies suggest that tea may protect against oxidative stress that induced tissue damage. A potential mechanism fr such effect y polyphenolc antioxidant tannic acid derived from tea was evaluated in tis study. Treatment of rats with ALCL3 for 80 days caused moderate toxicity on liver, kidney and splen as evidenced by increased LPO& No and a reduction of SOD,CAT, GST, GSH nd vitamin E.As well as histopathological changes.However improvement was noticed in the rats treated with tannic acid.

During the late pregnancy and during parturition in bovine, sevearl hormones and reoxygenation of placental tissues and consequences oxidative stress were involved. In this study blood samples from pregnant cows in the last three weeks of pregnancy were taken. At parturition 10 animals were selected as normal parturition and non-retaind placental expulsion. Other 10 cows were selected with normal parturition and retaind placenta. Another blood samples were taken after 1, 24 and 48 hours postpartum from both the obivois groups. The study concluded that oxidative stress and cortisol hormone might play an important role in the process of parturition in cows. It was also found that the extra-elevation of the LPO, NO and cortisol and the reduction of antioxidants could be used as indictor for the possibility of retention of fetal membrane after parturition.