The level of natural radioactivity in rocks and soil of 32 samples collected from locations at North Sana'a in Yemen was measured.
Concentrations of radionuclides in rocks and soils samples were determined by gamma-ray spectrometer using high purity
germanium (HPGe) detector with specially designed shield. The average radioactivity concentrations of 226Ra, 232Th, 40K were
determined and expressed in Bq/kg. The results showed that these radionuclides were present in concentrations of 21.79 ±
3.1, 19.5 ± 2.6 and 399.3 ± 16 Bq/kg, respectively, for rocks. For soil, the corresponding values were 48.2 ± 4.4, 41.7 ± 4.5
and 939.1 ± 36 Bq/kg, respectively. Also, the radiological hazard of the natural radionuclide content, radium equivalent activity,
total dose rates, external hazard index and gamma activity concentration index of the (rocks/soils) samples in the area under
consideration were calculated. The dose rates at 1 m above the ground from terrestrial sources were 38.39 and 86.89 nGy/h
for rocks and surface soil, respectively, which present no significant health hazards to humans.

The natural radioactivities of 40K, 226Ra, and 232Th and the fallout of 137Cs in rock and soil samples collected
around Juban town in Yemen (south west of Asia) were measured. Concentrations of radionuclides in
samples were determined by gamma-ray spectrometer using HPGe detector with specially designed shield.
The average radioactivity concentrations of 226Ra, 232Th, and 40K were determined expressed in Bq/kg. The
results show that these radionuclides were present in concentrations of (53.674, 12776.7, and
1742.8762 Bq/kg), (5574, 12176.6, and 2341778 Bq/kg), (212.878.7, 109 75.5, and 32.474.7 Bq/kg),
and (32.173, 22.372.9 and 190.9715 Bq/kg) for granite, gneiss, siltstone, and sandstone rocks,
respectively. For soil the corresponding values were 44.474.5, 58.275.1, and 822.7731 Bq/kg. Low
deposits of 137Cs were noted in investigation area, where the activity concentrations ranged from 0.170.1
to 23.271.2 Bq/kg. Also the radiological hazard of the natural radionuclides content, radium equivalent
activity, total dose rates, external hazard index, and gamma activity concentration index of the (rocks/soils)
samples in the area under consideration were calculated. The data were discussed and compared with
those given in the literature.

A series of coordination polymers of the general formula {[M(BDC)(azoles)(H2 O)m].xH2O}n (where
M = Co(II), Ni(II), and Cu(II); BDC = 1,4-benzenedicarboxylate; azoles = 2-aminobenzothiazole, 2-
aminothiazole, and 2-amino-4-methyl-thiazole; m = 0 or 1; and x = 1 or 2) were prepared and characterized.
The complexes were characterized based on elemental analysis, infrared and electronic spectral studies, magnetic
measurements, molar conductance, thermal analysis, X-ray diffraction, scanning electron microscopy,
and biological activity. Thermogravimetry, derivative thermogravimetry, and differential thermal analysis
were used to study the thermal decomposition of the complexes. The kinetic parameters were calculated
making use of the Coats-Redfern and Horowitz-Metzger equations.

NMOR can be formed endogenously from nitrite and morpholine. Oxidative damage may represent an important step in the toxicity of NMOR. This study was designed to evaluate the biological activity of 3-aminothiazolo[3.2a]benzimadzole-2-carbonitrile in the protection of hepatic and renal tissues against oxidative stress that is induced by administration of nitrite and morpholine as food additives in drinking water for 15 weeks. Conclusions: Nitrite plus morpholine administration to rats results in oxidative damage in hepatic and renal tissues. This damage may return to the increased production of ROS and changes in the levels of antioxidants. This oxidative damage was ameliorated by DMSO and 3-aminothiazolo[3.2a]benzimadzole-2-carbonitrile co-treatment.

NMOR can be formed endogenously from nitrite and morpholine. Oxidative damage may represent an important step in the toxicity of NMOR. This study was designed to evaluate the biological activity of 3-aminothiazolo[3.2a]benzimadzole-2-carbonitrile in the protection of hepatic and renal tissues against oxidative stress that is induced by administration of nitrite and morpholine as food additives in drinking water for 15 weeks. Conclusions: Nitrite plus morpholine administration to rats results in oxidative damage in hepatic and renal tissues. This damage may return to the increased production of ROS and changes in the levels of antioxidants. This oxidative damage was ameliorated by DMSO and 3-aminothiazolo[3.2a]benzimadzole-2-carbonitrile co-treatment.

NMOR can be formed endogenously from nitrite and morpholine. Oxidative damage may represent an important step in the toxicity of NMOR. This study was designed to evaluate the biological activity of 3-aminothiazolo[3.2a]benzimadzole-2-carbonitrile in the protection of hepatic and renal tissues against oxidative stress that is induced by administration of nitrite and morpholine as food additives in drinking water for 15 weeks. Conclusions: Nitrite plus morpholine administration to rats results in oxidative damage in hepatic and renal tissues. This damage may return to the increased production of ROS and changes in the levels of antioxidants. This oxidative damage was ameliorated by DMSO and 3-aminothiazolo[3.2a]benzimadzole-2-carbonitrile co-treatment.

The quantum equation of state of the electron-ion-plasmas in thermal equilibrium is derived; the pressure
until the third virial coefficient is calculated. We consider only the thermal equilibrium plasma in the case of
nλ3
ab  1 where λ2
ab = 2
mabKT is the thermal De Broglie wavelength. The formulas contain the contributions
of the exchange effects. Our equation of state is compared with result of Ebeling et al. who calculated the
pressure until the second virial coefficient only and in the case of χλee  1 where χ is the Debye length. Upon
comparison with Ebeling et al. one observes that the considerable contribution of the third virial coefficient in
the region of high temperatures.