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We study the dynamics of the intrinsic noise (IN) model of a three-level atom (qutrit) in V-configuration. The qutrit is inside an open cavity with a Kerr-like medium. The quantifiers of the atomic population, entanglement and mixedness are evaluated under the physical parameters of the photon multiplicity, IN and the nonlinearity of both the unitary interaction and the Kerr-like medium. It is found that the generated entanglement and mixedness may be enhanced by filling the cavity with a nonlinear medium.
The transfer of the energy between the two upper states and the lower state are very sensitive to the physical parameters. The entanglement and mixedness depend on the initial coherent intensity, the IN and the Kerr-like medium.

We study the dynamics of the intrinsic noise (IN) model of a three-level atom (qutrit) in V-configuration. The qutrit is inside an open cavity with a Kerr-like medium. The quantifiers of the atomic population, entanglement and mixedness are evaluated under the physical parameters of the photon multiplicity, IN and the nonlinearity of both the unitary interaction and the Kerr-like medium. It is found that the generated entanglement and mixedness may be enhanced by filling the cavity with a nonlinear medium.
The transfer of the energy between the two upper states and the lower state are very sensitive to the physical parameters. The entanglement and mixedness depend on the initial coherent intensity, the IN and the Kerr-like medium.

We study the dynamics of the intrinsic noise (IN) model of a three-level atom (qutrit) in V-configuration. The qutrit is inside an open cavity with a Kerr-like medium. The quantifiers of the atomic population, entanglement and mixedness are evaluated under the physical parameters of the photon multiplicity, IN and the nonlinearity of both the unitary interaction and the Kerr-like medium. It is found that the generated entanglement and mixedness may be enhanced by filling the cavity with a nonlinear medium.
The transfer of the energy between the two upper states and the lower state are very sensitive to the physical parameters. The entanglement and mixedness depend on the initial coherent intensity, the IN and the Kerr-like medium.

Members of family Acanthamoebidae and
Vahlkampfiidae are amphizoic, occurring as human
parasite causing many diseases. This study was aims
to evaluate the efficacy of ellagic acid (EA) to
ameloriate the histological changes in cornea and
the changes in oxidative stress markers in different
organs of rats infected with Allovahlkampfia
spelaea. Thirty rats were intraocular infected with
trophozites of A. spelaea. Fourteen days later, rats
divided into four groups, treated with chlorhexidine
(CHX), EA, EA plus CHX, and lubricant eye drops
for 14 days, respectively. All eyes were examined
clinically then rats were killed and their corneas,
brain, liver, ling, kidney and spleen were excised
and used for histological and biochemicals
evaluation. Eyes from A. spelaea infected rats
showed corneal ulcer with disruption of corneal
layer, congestion and infilteration of the inflammatory
cell in stromal layer. However, the cornea
of CHX, EA, and the combination of CHX and EA
treated rats showed hyperplasia in the epithelial
layer of cornea, hyperplasia in the epithelial layer of
cornea with stromal vascularization and epithelial
hyperplasia, stromal vascularization with fibroblast
cell activation, respectively. The activity of
acetylchoinestase in the brain and the markers of
oxidative stress in the brain, lung, liver, kidney and
spleen were altered in infected rats with A. spelaea
and restored by treatment with CHX, EA, or the
combination of CHX and EA. In conclusion
infection with A. spelaea induced keratitis and
biochemical changes in organs of rats, these changes
were ameloriated by the treatment with CHX or EA
or the combination of both with the priority of the
later one.

Members of family Acanthamoebidae and
Vahlkampfiidae are amphizoic, occurring as human
parasite causing many diseases. This study was aims
to evaluate the efficacy of ellagic acid (EA) to
ameloriate the histological changes in cornea and
the changes in oxidative stress markers in different
organs of rats infected with Allovahlkampfia
spelaea. Thirty rats were intraocular infected with
trophozites of A. spelaea. Fourteen days later, rats
divided into four groups, treated with chlorhexidine
(CHX), EA, EA plus CHX, and lubricant eye drops
for 14 days, respectively. All eyes were examined
clinically then rats were killed and their corneas,
brain, liver, ling, kidney and spleen were excised
and used for histological and biochemicals
evaluation. Eyes from A. spelaea infected rats
showed corneal ulcer with disruption of corneal
layer, congestion and infilteration of the inflammatory
cell in stromal layer. However, the cornea
of CHX, EA, and the combination of CHX and EA
treated rats showed hyperplasia in the epithelial
layer of cornea, hyperplasia in the epithelial layer of
cornea with stromal vascularization and epithelial
hyperplasia, stromal vascularization with fibroblast
cell activation, respectively. The activity of
acetylchoinestase in the brain and the markers of
oxidative stress in the brain, lung, liver, kidney and
spleen were altered in infected rats with A. spelaea
and restored by treatment with CHX, EA, or the
combination of CHX and EA. In conclusion
infection with A. spelaea induced keratitis and
biochemical changes in organs of rats, these changes
were ameloriated by the treatment with CHX or EA
or the combination of both with the priority of the
later one.

Members of family Acanthamoebidae and
Vahlkampfiidae are amphizoic, occurring as human
parasite causing many diseases. This study was aims
to evaluate the efficacy of ellagic acid (EA) to
ameloriate the histological changes in cornea and
the changes in oxidative stress markers in different
organs of rats infected with Allovahlkampfia
spelaea. Thirty rats were intraocular infected with
trophozites of A. spelaea. Fourteen days later, rats
divided into four groups, treated with chlorhexidine
(CHX), EA, EA plus CHX, and lubricant eye drops
for 14 days, respectively. All eyes were examined
clinically then rats were killed and their corneas,
brain, liver, ling, kidney and spleen were excised
and used for histological and biochemicals
evaluation. Eyes from A. spelaea infected rats
showed corneal ulcer with disruption of corneal
layer, congestion and infilteration of the inflammatory
cell in stromal layer. However, the cornea
of CHX, EA, and the combination of CHX and EA
treated rats showed hyperplasia in the epithelial
layer of cornea, hyperplasia in the epithelial layer of
cornea with stromal vascularization and epithelial
hyperplasia, stromal vascularization with fibroblast
cell activation, respectively. The activity of
acetylchoinestase in the brain and the markers of
oxidative stress in the brain, lung, liver, kidney and
spleen were altered in infected rats with A. spelaea
and restored by treatment with CHX, EA, or the
combination of CHX and EA. In conclusion
infection with A. spelaea induced keratitis and
biochemical changes in organs of rats, these changes
were ameloriated by the treatment with CHX or EA
or the combination of both with the priority of the
later one.

Members of family Acanthamoebidae and
Vahlkampfiidae are amphizoic, occurring as human
parasite causing many diseases. This study was aims
to evaluate the efficacy of ellagic acid (EA) to
ameloriate the histological changes in cornea and
the changes in oxidative stress markers in different
organs of rats infected with Allovahlkampfia
spelaea. Thirty rats were intraocular infected with
trophozites of A. spelaea. Fourteen days later, rats
divided into four groups, treated with chlorhexidine
(CHX), EA, EA plus CHX, and lubricant eye drops
for 14 days, respectively. All eyes were examined
clinically then rats were killed and their corneas,
brain, liver, ling, kidney and spleen were excised
and used for histological and biochemicals
evaluation. Eyes from A. spelaea infected rats
showed corneal ulcer with disruption of corneal
layer, congestion and infilteration of the inflammatory
cell in stromal layer. However, the cornea
of CHX, EA, and the combination of CHX and EA
treated rats showed hyperplasia in the epithelial
layer of cornea, hyperplasia in the epithelial layer of
cornea with stromal vascularization and epithelial
hyperplasia, stromal vascularization with fibroblast
cell activation, respectively. The activity of
acetylchoinestase in the brain and the markers of
oxidative stress in the brain, lung, liver, kidney and
spleen were altered in infected rats with A. spelaea
and restored by treatment with CHX, EA, or the
combination of CHX and EA. In conclusion
infection with A. spelaea induced keratitis and
biochemical changes in organs of rats, these changes
were ameloriated by the treatment with CHX or EA
or the combination of both with the priority of the
later one.

Members of family Acanthamoebidae and
Vahlkampfiidae are amphizoic, occurring as human
parasite causing many diseases. This study was aims
to evaluate the efficacy of ellagic acid (EA) to
ameloriate the histological changes in cornea and
the changes in oxidative stress markers in different
organs of rats infected with Allovahlkampfia
spelaea. Thirty rats were intraocular infected with
trophozites of A. spelaea. Fourteen days later, rats
divided into four groups, treated with chlorhexidine
(CHX), EA, EA plus CHX, and lubricant eye drops
for 14 days, respectively. All eyes were examined
clinically then rats were killed and their corneas,
brain, liver, ling, kidney and spleen were excised
and used for histological and biochemicals
evaluation. Eyes from A. spelaea infected rats
showed corneal ulcer with disruption of corneal
layer, congestion and infilteration of the inflammatory
cell in stromal layer. However, the cornea
of CHX, EA, and the combination of CHX and EA
treated rats showed hyperplasia in the epithelial
layer of cornea, hyperplasia in the epithelial layer of
cornea with stromal vascularization and epithelial
hyperplasia, stromal vascularization with fibroblast
cell activation, respectively. The activity of
acetylchoinestase in the brain and the markers of
oxidative stress in the brain, lung, liver, kidney and
spleen were altered in infected rats with A. spelaea
and restored by treatment with CHX, EA, or the
combination of CHX and EA. In conclusion
infection with A. spelaea induced keratitis and
biochemical changes in organs of rats, these changes
were ameloriated by the treatment with CHX or EA
or the combination of both with the priority of the
later one.

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