A series of coordination polymers of the general formula {[M(BDC)(azoles)(H2 O)m].xH2O}n (where M = Co(II), Ni(II), and Cu(II); BDC = 1,4-benzenedicarboxylate; azoles = 2-aminobenzothiazole, 2- aminothiazole, and 2-amino-4-methyl-thiazole; m = 0 or 1; and x = 1 or 2) were prepared and characterized. The complexes were characterized based on elemental analysis, infrared and electronic spectral studies, magnetic measurements, molar conductance, thermal analysis, X-ray diffraction, scanning electron microscopy, and biological activity. Thermogravimetry, derivative thermogravimetry, and differential thermal analysis were used to study the thermal decomposition of the complexes. The kinetic parameters were calculated making use of the Coats-Redfern and Horowitz-Metzger equations.

NMOR can be formed endogenously from nitrite and morpholine. Oxidative damage may represent an important step in the toxicity of NMOR. This study was designed to evaluate the biological activity of 3-aminothiazolo[3.2a]benzimadzole-2-carbonitrile in the protection of hepatic and renal tissues against oxidative stress that is induced by administration of nitrite and morpholine as food additives in drinking water for 15 weeks. Conclusions: Nitrite plus morpholine administration to rats results in oxidative damage in hepatic and renal tissues. This damage may return to the increased production of ROS and changes in the levels of antioxidants. This oxidative damage was ameliorated by DMSO and 3-aminothiazolo[3.2a]benzimadzole-2-carbonitrile co-treatment.

NMOR can be formed endogenously from nitrite and morpholine. Oxidative damage may represent an important step in the toxicity of NMOR. This study was designed to evaluate the biological activity of 3-aminothiazolo[3.2a]benzimadzole-2-carbonitrile in the protection of hepatic and renal tissues against oxidative stress that is induced by administration of nitrite and morpholine as food additives in drinking water for 15 weeks. Conclusions: Nitrite plus morpholine administration to rats results in oxidative damage in hepatic and renal tissues. This damage may return to the increased production of ROS and changes in the levels of antioxidants. This oxidative damage was ameliorated by DMSO and 3-aminothiazolo[3.2a]benzimadzole-2-carbonitrile co-treatment.

NMOR can be formed endogenously from nitrite and morpholine. Oxidative damage may represent an important step in the toxicity of NMOR. This study was designed to evaluate the biological activity of 3-aminothiazolo[3.2a]benzimadzole-2-carbonitrile in the protection of hepatic and renal tissues against oxidative stress that is induced by administration of nitrite and morpholine as food additives in drinking water for 15 weeks. Conclusions: Nitrite plus morpholine administration to rats results in oxidative damage in hepatic and renal tissues. This damage may return to the increased production of ROS and changes in the levels of antioxidants. This oxidative damage was ameliorated by DMSO and 3-aminothiazolo[3.2a]benzimadzole-2-carbonitrile co-treatment.

The quantum equation of state of the electron-ion-plasmas in thermal equilibrium is derived; the pressure until the third virial coefficient is calculated. We consider only the thermal equilibrium plasma in the case of nλ3 ab  1 where λ2 ab = 2 mabKT is the thermal De Broglie wavelength. The formulas contain the contributions of the exchange effects. Our equation of state is compared with result of Ebeling et al. who calculated the pressure until the second virial coefficient only and in the case of χλee  1 where χ is the Debye length. Upon comparison with Ebeling et al. one observes that the considerable contribution of the third virial coefficient in the region of high temperatures.