Chalcogenide Se50Te20S30 thin film of different thickness was deposited using thermal evaporation technique. The thermogram of the chalcogenide bulk Se50- Te20S30 was obtained using a differential scanning calorimetry (DSC) with heating rate of 7.5 K/min. The glass transition temperature Tg, crystallization temperature Tc and peak crystallization temperature Tp were identified. The X-ray diffraction (XRD) examination indicates the amorphous nature of the as-deposited film and polycrystalline structure of the thermal annealed ones. The dark electrical resistivity (q) measurements were taken in temperature range (300–500 K) and thickness range (200–450 nm). Analysis of the electrical resistivity results revealed two types of conduction mechanisms: conduction due to extended states in the temperature range (T[Tc) and variable range hopping in the temperature range (TTc). The effect of the heat treatment and thickness on the density of localized states at the Fermi level N(EF) and hopping parameters were studied.

Type I diabetes (T1D) is a characterized by the inflammation of pancreatic islets and destruction of β cells. Long and persistent uncontrolled diabetes tends to degenerate the immune system and increase the incidence of infections in diabetic individuals. Most serious diabetic complications are mediated by the free radicals, which damage multiple cellular components through direct effects of the cell cycle regulatory proteins. Camel whey protein (CWP) has antioxidant activity and decreases the effects of free radicals. However, the effects of CWP on lymphoid organs have not been studied in the context of diabetes. Therefore, the present study was designed to investigate the dietary influence of CWP supplementation on the lymphoid organs in streptozotocin (STZ)-induced type 1 diabetic mouse model. Three experimental groups were used: non diabetic control mice, diabetic mice, and diabetic mice treated with CWP. Induction of diabetes was associated with a marked reduction in glutathione (GSH) levels; decreased activities of GSH peroxidase (GSH Px), manganese superoxide dismutase (MnSOD) and catalase; increased reactive oxygen species (ROS) levels and iNOS activity in plasma and lymphoid organs. Furthermore, diabetic mice exhibited alterations in the expression of Bax and Bcl-XL, and subsequently pathological alterations in the architecture of the bone marrow, pancreas, thymus, and spleen. Interestingly, treatment of diabetic mice with CWP robustly restored glucose, insulin, GSH, and ROS levels and the activities of GSH Px, MnSOD, catalase and iNOS. Additionally, supplementation of diabetic mice with CWP improvement in the architecture of lymphoid tissues and rescued from apoptosis through direct effects on the Bax and Bcl-XL proteins. These data revealed the therapeutic potential of CWP against diabetic complications mediated damages of lymphoid organs.

Type I diabetes (T1D) is a characterized by the inflammation of pancreatic islets and destruction of β cells. Long and persistent uncontrolled diabetes tends to degenerate the immune system and increase the incidence of infections in diabetic individuals. Most serious diabetic complications are mediated by the free radicals, which damage multiple cellular components through direct effects of the cell cycle regulatory proteins. Camel whey protein (CWP) has antioxidant activity and decreases the effects of free radicals. However, the effects of CWP on lymphoid organs have not been studied in the context of diabetes. Therefore, the present study was designed to investigate the dietary influence of CWP supplementation on the lymphoid organs in streptozotocin (STZ)-induced type 1 diabetic mouse model. Three experimental groups were used: non diabetic control mice, diabetic mice, and diabetic mice treated with CWP. Induction of diabetes was associated with a marked reduction in glutathione (GSH) levels; decreased activities of GSH peroxidase (GSH Px), manganese superoxide dismutase (MnSOD) and catalase; increased reactive oxygen species (ROS) levels and iNOS activity in plasma and lymphoid organs. Furthermore, diabetic mice exhibited alterations in the expression of Bax and Bcl-XL, and subsequently pathological alterations in the architecture of the bone marrow, pancreas, thymus, and spleen. Interestingly, treatment of diabetic mice with CWP robustly restored glucose, insulin, GSH, and ROS levels and the activities of GSH Px, MnSOD, catalase and iNOS. Additionally, supplementation of diabetic mice with CWP improvement in the architecture of lymphoid tissues and rescued from apoptosis through direct effects on the Bax and Bcl-XL proteins. These data revealed the therapeutic potential of CWP against diabetic complications mediated damages of lymphoid organs.

Type I diabetes (T1D) is a characterized by the inflammation of pancreatic islets and destruction of β cells. Long and persistent uncontrolled diabetes tends to degenerate the immune system and increase the incidence of infections in diabetic individuals. Most serious diabetic complications are mediated by the free radicals, which damage multiple cellular components through direct effects of the cell cycle regulatory proteins. Camel whey protein (CWP) has antioxidant activity and decreases the effects of free radicals. However, the effects of CWP on lymphoid organs have not been studied in the context of diabetes. Therefore, the present study was designed to investigate the dietary influence of CWP supplementation on the lymphoid organs in streptozotocin (STZ)-induced type 1 diabetic mouse model. Three experimental groups were used: non diabetic control mice, diabetic mice, and diabetic mice treated with CWP. Induction of diabetes was associated with a marked reduction in glutathione (GSH) levels; decreased activities of GSH peroxidase (GSH Px), manganese superoxide dismutase (MnSOD) and catalase; increased reactive oxygen species (ROS) levels and iNOS activity in plasma and lymphoid organs. Furthermore, diabetic mice exhibited alterations in the expression of Bax and Bcl-XL, and subsequently pathological alterations in the architecture of the bone marrow, pancreas, thymus, and spleen. Interestingly, treatment of diabetic mice with CWP robustly restored glucose, insulin, GSH, and ROS levels and the activities of GSH Px, MnSOD, catalase and iNOS. Additionally, supplementation of diabetic mice with CWP improvement in the architecture of lymphoid tissues and rescued from apoptosis through direct effects on the Bax and Bcl-XL proteins. These data revealed the therapeutic potential of CWP against diabetic complications mediated damages of lymphoid organs.

Type I diabetes (T1D) is a characterized by the inflammation of pancreatic islets and destruction of β cells. Long and persistent uncontrolled diabetes tends to degenerate the immune system and increase the incidence of infections in diabetic individuals. Most serious diabetic complications are mediated by the free radicals, which damage multiple cellular components through direct effects of the cell cycle regulatory proteins. Camel whey protein (CWP) has antioxidant activity and decreases the effects of free radicals. However, the effects of CWP on lymphoid organs have not been studied in the context of diabetes. Therefore, the present study was designed to investigate the dietary influence of CWP supplementation on the lymphoid organs in streptozotocin (STZ)-induced type 1 diabetic mouse model. Three experimental groups were used: non diabetic control mice, diabetic mice, and diabetic mice treated with CWP. Induction of diabetes was associated with a marked reduction in glutathione (GSH) levels; decreased activities of GSH peroxidase (GSH Px), manganese superoxide dismutase (MnSOD) and catalase; increased reactive oxygen species (ROS) levels and iNOS activity in plasma and lymphoid organs. Furthermore, diabetic mice exhibited alterations in the expression of Bax and Bcl-XL, and subsequently pathological alterations in the architecture of the bone marrow, pancreas, thymus, and spleen. Interestingly, treatment of diabetic mice with CWP robustly restored glucose, insulin, GSH, and ROS levels and the activities of GSH Px, MnSOD, catalase and iNOS. Additionally, supplementation of diabetic mice with CWP improvement in the architecture of lymphoid tissues and rescued from apoptosis through direct effects on the Bax and Bcl-XL proteins. These data revealed the therapeutic potential of CWP against diabetic complications mediated damages of lymphoid organs.

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A series of single and mixed oxide nanocatalysts of mesoporous CuO-Fe2O3with different CuO contents(1–50 wt.%) were prepared by a co-precipitation method and further promoted by trace amounts ofCeO2, ZrO2and Ag2O (0.1–0.5 wt.%) dopants. The original and calcined catalysts were characterized byTG, DTA, XRD, TEM, VSM, N2sorption analysis, surface chemisorbed oxygen and DC electrical conductivitymeasurements. The catalytic performance of these nanocatalysts toward CO oxidation was studied using aconventional fixed bed flow type reactor. The results revealed that the addition of 1–20 wt.% CuO to Fe2O3monotonically increases the specific surface area, the amount of surface chemisorbed oxygen, electricalconductivity and catalytic activity of the nanocatalysts. In addition, the catalytic activity indicated thatFe-Cu mixed oxide nanocatalyst promoted with the three dopants (CeO2, ZrO2and Ag2O) exhibited thehighest catalytic activity with a total conversion of CO into CO2at 100◦C. Moreover, the activation energyof CO oxidation decreased from 38.4 to 23.1 kJmol−1upon treating the catalyst containing 20 wt.% CuOwith the three dopants. Finally the effects of various operational parameters were also studied.

A series of single and mixed oxide nanocatalysts of mesoporous CuO-Fe2O3with different CuO contents(1–50 wt.%) were prepared by a co-precipitation method and further promoted by trace amounts ofCeO2, ZrO2and Ag2O (0.1–0.5 wt.%) dopants. The original and calcined catalysts were characterized byTG, DTA, XRD, TEM, VSM, N2sorption analysis, surface chemisorbed oxygen and DC electrical conductivitymeasurements. The catalytic performance of these nanocatalysts toward CO oxidation was studied using aconventional fixed bed flow type reactor. The results revealed that the addition of 1–20 wt.% CuO to Fe2O3monotonically increases the specific surface area, the amount of surface chemisorbed oxygen, electricalconductivity and catalytic activity of the nanocatalysts. In addition, the catalytic activity indicated thatFe-Cu mixed oxide nanocatalyst promoted with the three dopants (CeO2, ZrO2and Ag2O) exhibited thehighest catalytic activity with a total conversion of CO into CO2at 100◦C. Moreover, the activation energyof CO oxidation decreased from 38.4 to 23.1 kJmol−1upon treating the catalyst containing 20 wt.% CuOwith the three dopants. Finally the effects of various operational parameters were also studied.

A series of single and mixed oxide nanocatalysts of mesoporous CuO-Fe2O3with different CuO contents(1–50 wt.%) were prepared by a co-precipitation method and further promoted by trace amounts ofCeO2, ZrO2and Ag2O (0.1–0.5 wt.%) dopants. The original and calcined catalysts were characterized byTG, DTA, XRD, TEM, VSM, N2sorption analysis, surface chemisorbed oxygen and DC electrical conductivitymeasurements. The catalytic performance of these nanocatalysts toward CO oxidation was studied using aconventional fixed bed flow type reactor. The results revealed that the addition of 1–20 wt.% CuO to Fe2O3monotonically increases the specific surface area, the amount of surface chemisorbed oxygen, electricalconductivity and catalytic activity of the nanocatalysts. In addition, the catalytic activity indicated thatFe-Cu mixed oxide nanocatalyst promoted with the three dopants (CeO2, ZrO2and Ag2O) exhibited thehighest catalytic activity with a total conversion of CO into CO2at 100◦C. Moreover, the activation energyof CO oxidation decreased from 38.4 to 23.1 kJmol−1upon treating the catalyst containing 20 wt.% CuOwith the three dopants. Finally the effects of various operational parameters were also studied.

tThe catalytic oxidation of CO into CO2on mesoporous Fe–Co mixed oxide nanocatalystsat low temperature was carried out. The catalysts with different ratios of Co3O4(1–30 wt.%)were prepared by a simple co-precipitation method. The original and calcined catalysts werecharacterized by TG, DTA, XRD, TEM, VSM, N2sorption analysis, surface chemisorbed oxy-gen and dc electrical conductivity measurements. The results revealed that the addition ofCo3O4to Fe2O3monotonically increases the amount of surface chemisorbed oxygen, elec-trical conductivity and catalytic activity of the nanocatalysts. The role of the active redoxsites established in these nanocatalysts such as, Co3+/Co2+, Fe3+/Fe2+and Co3+/Fe2+whichare responsible for such modification was discussed. The magnetic studies indicated thatthe Fe–Co mixed oxide nanocatalysts exhibited ferromagnetic nature and the catalyst con-taining 30 wt.% Co3O4calcined at 600◦C possessed the highest saturation magnetization(Ms= 51.5 emu g−1). In addition the kinetic data illustrated that, the activation energy val-ues of CO oxidation gradually decreased with increasing of Co3O4content. Moreover, thecatalytic behavior under different atmospheres during calcination was also studied.