The glass transition and the crystallization kinetics of Zn10Se90 glass by differential thermal analys is (DTA) technique under non-isothermal conduction were studied. The effective activation energies of the glass transition and the crystallization have been
evaluated on the basses of the Kissinger and Matusita et al. approximations. Kineticparameters of the crystallization process are significantly influenced by the heating rate. We have compared the experimental DTA with the calculated data curves for
Zn10Se90 system using the Johanson-Mehl-Avrami (JMA) and Sestak-Berggren SB(M,N) models. Simulation results indicated that, the SB(M,N) model is more
suitable for describing the crystallization kinetics for the studied composition. Furthermore, the crystalline phases of annealed Zn10Se90 were characterized by X-ray diffraction. The surface morphology of the annealed samples was examined using scanning electron microscopy (SEM).

The glass transition and the crystallization kinetics of Zn10Se90 glass by differential thermal analys is (DTA) technique under non-isothermal conduction were studied. The effective activation energies of the glass transition and the crystallization have been
evaluated on the basses of the Kissinger and Matusita et al. approximations. Kineticparameters of the crystallization process are significantly influenced by the heating rate. We have compared the experimental DTA with the calculated data curves for
Zn10Se90 system using the Johanson-Mehl-Avrami (JMA) and Sestak-Berggren SB(M,N) models. Simulation results indicated that, the SB(M,N) model is more
suitable for describing the crystallization kinetics for the studied composition. Furthermore, the crystalline phases of annealed Zn10Se90 were characterized by X-ray diffraction. The surface morphology of the annealed samples was examined using scanning electron microscopy (SEM).

The glass transition and the crystallization kinetics of Zn10Se90 glass by differential thermal analys is (DTA) technique under non-isothermal conduction were studied. The effective activation energies of the glass transition and the crystallization have been
evaluated on the basses of the Kissinger and Matusita et al. approximations. Kineticparameters of the crystallization process are significantly influenced by the heating rate. We have compared the experimental DTA with the calculated data curves for
Zn10Se90 system using the Johanson-Mehl-Avrami (JMA) and Sestak-Berggren SB(M,N) models. Simulation results indicated that, the SB(M,N) model is more
suitable for describing the crystallization kinetics for the studied composition. Furthermore, the crystalline phases of annealed Zn10Se90 were characterized by X-ray diffraction. The surface morphology of the annealed samples was examined using scanning electron microscopy (SEM).

ABSTRACT
The effect of nutrients deficiency (phosphorus, nitrogen and
sulphur) on growth, ultrastructure, β-carotene and total lipid contents of
the green alga Asteromonas gracilis has been examined. Algal cells were
cultured under conditions of nutrients deficiency. The maximum growth
rate and minimum generation time calculated on basis of chl. a were
0.032 and 21.9, respectively for control culture. The most changes in
ultrastructure of A. gracilis observed when cells were cultivated without
nitrogen source, the changes are in the structure of chloroplast, where the
degradation of thylakoids was observed and the grana structure of
thylakoids had almost disappeared as well as accumulation of starch
grains were easily detected. The total carbohydrates were decreased with
deficiency of phosphorus and sulphur, but increased in case of nitrogen
deficiency. On the other hand, the production of β-carotene and total
lipids was enhanced with phosphorus, nitrogen and sulphur deficiency.

ABSTRACT
The effect of nutrients deficiency (phosphorus, nitrogen and
sulphur) on growth, ultrastructure, β-carotene and total lipid contents of
the green alga Asteromonas gracilis has been examined. Algal cells were
cultured under conditions of nutrients deficiency. The maximum growth
rate and minimum generation time calculated on basis of chl. a were
0.032 and 21.9, respectively for control culture. The most changes in
ultrastructure of A. gracilis observed when cells were cultivated without
nitrogen source, the changes are in the structure of chloroplast, where the
degradation of thylakoids was observed and the grana structure of
thylakoids had almost disappeared as well as accumulation of starch
grains were easily detected. The total carbohydrates were decreased with
deficiency of phosphorus and sulphur, but increased in case of nitrogen
deficiency. On the other hand, the production of β-carotene and total
lipids was enhanced with phosphorus, nitrogen and sulphur deficiency.

ABSTRACT
The effect of nutrients deficiency (phosphorus, nitrogen and
sulphur) on growth, ultrastructure, β-carotene and total lipid contents of
the green alga Asteromonas gracilis has been examined. Algal cells were
cultured under conditions of nutrients deficiency. The maximum growth
rate and minimum generation time calculated on basis of chl. a were
0.032 and 21.9, respectively for control culture. The most changes in
ultrastructure of A. gracilis observed when cells were cultivated without
nitrogen source, the changes are in the structure of chloroplast, where the
degradation of thylakoids was observed and the grana structure of
thylakoids had almost disappeared as well as accumulation of starch
grains were easily detected. The total carbohydrates were decreased with
deficiency of phosphorus and sulphur, but increased in case of nitrogen
deficiency. On the other hand, the production of β-carotene and total
lipids was enhanced with phosphorus, nitrogen and sulphur deficiency.

ABSTRACT
The effect of nutrients deficiency (phosphorus, nitrogen and
sulphur) on growth, ultrastructure, β-carotene and total lipid contents of
the green alga Asteromonas gracilis has been examined. Algal cells were
cultured under conditions of nutrients deficiency. The maximum growth
rate and minimum generation time calculated on basis of chl. a were
0.032 and 21.9, respectively for control culture. The most changes in
ultrastructure of A. gracilis observed when cells were cultivated without
nitrogen source, the changes are in the structure of chloroplast, where the
degradation of thylakoids was observed and the grana structure of
thylakoids had almost disappeared as well as accumulation of starch
grains were easily detected. The total carbohydrates were decreased with
deficiency of phosphorus and sulphur, but increased in case of nitrogen
deficiency. On the other hand, the production of β-carotene and total
lipids was enhanced with phosphorus, nitrogen and sulphur deficiency.

ABSTRACT
This study dealt with the histopathological changes in the lungs, liver, small intestine, muscles, kidneys and brain of mice experimentally infected with Toxascaris leonina. Larvae were found on the 8th dpi (day post infection) in the liver. Also, they observed on the 4th dpi in the small intestine, 8th dpi in lungs, but it could not be seen in both kidneys and brain. In muscles, the larvae were detected after one week of infection. The histopathological changes were observed in the form of infiltration with lymphocytes, eosinophils, with few areas of hemorrhages indicating the migratory pathway of the larvae. Moreover, the main features in the portal spaces in the lungs were the presence of edema, hemorrhages and diffuse infiltration of alveolar wall which appeared thickened as a result of interstitial edema and vascular congestion.
The histopathological results were in high accordance with the biochemical results. At 10th dpi, there were significant rises in the parameters of oxidative damage to lungs with the highest extent (lipid peroxidation LPO, nitric oxide NO and total peroxides TP) followed by muscles (LPO) and liver (NO). Also, some antioxidants in infected mice showed significant decrease in liver (catalase activity) and muscles (glutathione, GSH). Kidneys did not show any biochemical change. It could be concluded that Toxascaris leonina infection induces histopathological changes and oxidative damage to some organs of mice especially the lungs, muscles and small intestine. Also, these changes may reflect the direct relationship between the severity of infection with larvae and the degree of oxidative damage.

ABSTRACT
This study dealt with the histopathological changes in the lungs, liver, small intestine, muscles, kidneys and brain of mice experimentally infected with Toxascaris leonina. Larvae were found on the 8th dpi (day post infection) in the liver. Also, they observed on the 4th dpi in the small intestine, 8th dpi in lungs, but it could not be seen in both kidneys and brain. In muscles, the larvae were detected after one week of infection. The histopathological changes were observed in the form of infiltration with lymphocytes, eosinophils, with few areas of hemorrhages indicating the migratory pathway of the larvae. Moreover, the main features in the portal spaces in the lungs were the presence of edema, hemorrhages and diffuse infiltration of alveolar wall which appeared thickened as a result of interstitial edema and vascular congestion.
The histopathological results were in high accordance with the biochemical results. At 10th dpi, there were significant rises in the parameters of oxidative damage to lungs with the highest extent (lipid peroxidation LPO, nitric oxide NO and total peroxides TP) followed by muscles (LPO) and liver (NO). Also, some antioxidants in infected mice showed significant decrease in liver (catalase activity) and muscles (glutathione, GSH). Kidneys did not show any biochemical change. It could be concluded that Toxascaris leonina infection induces histopathological changes and oxidative damage to some organs of mice especially the lungs, muscles and small intestine. Also, these changes may reflect the direct relationship between the severity of infection with larvae and the degree of oxidative damage.

Abstract
This study was designed to investigate the effect of Neem seed oil on the oxidative stress in gills and erythrocytes (EC), after 1, 2 and 3 weeks (W) and to evaluate the antioxidant effect of lupine seed supplementation (LS) in the Nile tilapia Oreochromis niloticus. Two doses of Neem oil; 1/20 LC50:56 (NO1) and 1/10 LC50:112 PPM (NO2) and three periods of one, two and three weeks were used. Oxidative stress parameter, total peroxide (TP) and antioxidant enzyme activities, catalase (CAT) and superoxide dismutase (SOD) as well as glycemia were measured. NO2 markedly decreased CAT and SOD activities in gills at most periods (52 - 99% and 16 – 52 %, respectively) and in EC (22 - 62 and 48 – 63 %, respectively) in most periods, but NO1 had less effect. In turn, both doses significantly enhanced TP levels in gills and EC. Moreover, hyperglycemia was detected three weeks after control (NO) exposure. LS significantly curtailed or abolished the adverse effect of NO exposure on oxidative and antioxidant parameters in gills and EC. This may be attributed to the hypoglycemic effect of LS as well as its antioxidant efficiency. In conclusion, NO exerts oxidative damage in the fish and LS has an antioxidant potency.